Articles | Volume 10, issue 4
https://doi.org/10.5194/se-10-1283-2019
https://doi.org/10.5194/se-10-1283-2019
Research article
 | 
02 Aug 2019
Research article |  | 02 Aug 2019

Overprinting translational domains in passive margin salt basins: insights from analogue modelling

Zhiyuan Ge, Matthias Rosenau, Michael Warsitzka, and Rob L. Gawthorpe

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Cited articles

Adam, J. and Krezsek, C.: Basin-scale salt tectonic processes of the Laurentian Basin, Eastern Canada: insights from integrated regional 2-D seismic interpretation and 4D physical experiments, Geol. Soc. London Spec. Pub., 363, 331–360, 2012. 
Adam, J., Urai, J. L., Wieneke, B., Oncken, O., Pfeiffer, K., Kukowski, N., Lohrmann, J., Hoth, S., van der Zee, W., and Schmatz, J.: Shear localisation and strain distribution during tectonic faulting – new insights from granular-flow experiments and high-resolution optical image correlation techniques, J. Struct. Geol., 27, 283–301, 2005. 
Adam, J., Ge, Z., and Sanchez, M.: Post-rift salt tectonic evolution and key control factors of the Jequitinhonha deepwater fold belt, central Brazil passive margin: Insights from scaled physical experiments, Mar. Pet. Geol., 37, 70–100, 2012a. 
Adam, J., Ge, Z., and Sanchez, M.: Salt-structural styles and kinematic evolution of the Jequitinhonha deepwater fold belt, central Brazil passive margin, Mar. Pet. Geol., 37, 101–120, 2012b. 
Allen, J. and Beaumont, C.: Impact of inconsistent density scaling on physical analogue models of continental margin scale salt tectonics, J. Geophys. Res.-Sol. Ea., 117, B08103, https://doi.org/10.1029/2012JB009227, 2012. 
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Salt basins are important as they bear abundant natural resources and record valuable geological information. However, some models of salt basin evolution do not fully reconcile with natural examples. Using state-of-the-art analogue modelling, we investigate how a relatively stable area, the translational domain, occurs and gets overprinted in salt basins. The results suggest that that variation of sediment deposition is the key factor for overprinting the translational domain.